John Foley - Profile and Journalist Details
Find journalists that align with your industry, location and vision. Unlock John Foley's full journalist profile, including location, coverage topics, current employer, biography and preferences. Sign up today and start building journalist relationships that fuel your startup's growth.
Get connected with journalists today
John Foley
Verified
Editor, Science Signaling
Washington, D.C., United States
Beats
Travel, Outdoor Activities, Marine Life Conservation, Cocktails, Wildlife Conservation
Content
The kinase PLK1 promotes Hedgehog signaling-dependent resistance to the antiandrogen enzalutamide in metastatic prostate cancer
By Qiongsi Zhang, Aoi Sugiyama, John Foley Verified, Jia Peng| Science Magazine Verified Research ArticleCANCER THERAPYINTRODUCTIONProstate cancer is a leading cause of cancer-related mortality among men globally. Androgen receptor (AR) signaling is pivotal in the development and progression of prostate cancer. For advanced prostate cancer, patients undergo concurrent androgen deprivation therapy to reduce male hormone levels, either by inhibiting hormone production or blocking their function on prostate cancer cells.
Structural analysis reveals how tetrameric tyrosine-phosphorylated STAT1 is targeted by the rabies virus P-protein
By Qiongsi Zhang, Aoi Sugiyama, John Foley Verified, Miku Minami| Science Magazine Verified Published In Science SignalingVolume 18 | Issue 878March 2025Article versionsSubmission historyReceived: 2 August 2024Accepted: 28 February 2025PermissionsRequest permissions for this article. AcknowledgmentsWe thank N. Shimizu for assistance in SAXS data collection at Photon Factory.
The kinase PLK1 promotes Hedgehog signaling-dependent resistance to the antiandrogen enzalutamide in metastatic prostate cancer
By Qiongsi Zhang, Aoi Sugiyama, John Foley Verified, Jia Peng| Science Magazine Verified Research ArticleCANCER THERAPYINTRODUCTIONProstate cancer is a leading cause of cancer-related mortality among men globally. Androgen receptor (AR) signaling is pivotal in the development and progression of prostate cancer. For advanced prostate cancer, patients undergo concurrent androgen deprivation therapy to reduce male hormone levels, either by inhibiting hormone production or blocking their function on prostate cancer cells.
Company Info
Science Signaling