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Kirsty Minton
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Senior Editor, Nature Reviews Immunology
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By Kirsty Minton Verified| Nature Verified IgE specific for inhaled allergens mediates airway hypersensitivity in asthma, a chronic condition with intermittent symptoms driven by allergen re-exposure. However, IgE-expressing B cells fail to form memory B cells (MBCs) and long-lived IgE+ plasma cells are rare. In addition, systemic, allergen-specific IgE responses are absent in some individuals with asthma. Thus, how allergen-specific IgE responses in the lung are maintained long term is unclear. Nelson et al.
By Kirsty Minton Verified| Nature Verified Neutrophils are highly pro-inflammatory cells with well-established roles in pathogen clearance through phagocytosis, the release of antimicrobial factors and production of neutrophil extracellular traps. All these neutrophil functions can cause tissue damage and therefore must be tightly regulated. Hsu et al. report in Cell that as neutrophils age they actively release CD55+ vesicles that inhibit complement activation and thus help to resolve inflammation.
By Kirsty Minton Verified| Nature Verified IgE specific for inhaled allergens mediates airway hypersensitivity in asthma, a chronic condition with intermittent symptoms driven by allergen re-exposure. However, IgE-expressing B cells fail to form memory B cells (MBCs) and long-lived IgE+ plasma cells are rare. In addition, systemic, allergen-specific IgE responses are absent in some individuals with asthma. Thus, how allergen-specific IgE responses in the lung are maintained long term is unclear. Nelson et al.
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Nature Reviews Immunology
London, England, United Kingdom
+44 20 7833 4000