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Sebastian Köhler

Sebastian Köhler

Verified

Online Editor, Berliner Morgenpost

Baden-Württemberg

Final Covers

Conflict, Foreign policy, Royals , Defense, Europe, Foreign affairs, Diplomacy, United Kingdom, Terrorism, Politics , England, Migration , Military, Britain, General News, Veterans , Cyberwarfare, Human rights

Doesn’t Cover

I don't cover anything if it doesn't have to do with Fashion.

Journalist Type

Seniority Positions

Industries

Medium Formats

Content

Total articles 2466

  • Bcl-2 Oligomerizes Bax on the Mitochondrial Membrane Surface Preventing the Initial Stages of Apoptosis

    By Sebastian Köhler, Sophie E Ayscough, Luke Ashley Clifton, Jörgen Ådén| bioRxiv@ AbstractThe Bcl-2 family of proteins control mitochondrial outer membrane (MOM) pore formation, crucial to cellular clearance via apoptosis. However, the molecular principles by which opposing family members inhibit, mediate or promote MOM perforation remain elusive. Here, we demonstrate that cell-protecting Bcl-2 directly sequesters cell-killing Bax into a protein-protein complex at the membrane interface preventing Bax forming apoptotic pores.

    By Sebastian Köhler, Sophie E Ayscough, Luke Ashley Clifton, Jörgen Ådén · bioRxiv

    Jan. 27, 2025

  • Wie wird man Spinnen im Haus einfach und nachhaltig wieder los?

    By Sebastian Köhler| MSN Deutschland Verified Microsoft tritt für den Schutz Ihrer Privatsphäre ein. Microsoft und unsere Drittanbieter verwenden Cookies, um Informationen wie eindeutige IDs zu speichern und darauf zuzugreifen, um unsere Dienste und Anzeigen bereitzustellen, zu pflegen und zu verbessern. Wenn Sie zustimmen, personalisieren MSN und Microsoft Bing die angezeigten Inhalte und Anzeigen.

    By Sebastian Köhler · MSN Deutschland

    Jan. 22, 2025

  • Bcl-2 Oligomerizes Bax on the Mitochondrial Membrane Surface Preventing the Initial Stages of Apoptosis

    By Sebastian Köhler, Sophie E Ayscough, Luke Ashley Clifton, Jörgen Ådén| bioRxiv@ AbstractThe Bcl-2 family of proteins control mitochondrial outer membrane (MOM) pore formation, crucial to cellular clearance via apoptosis. However, the molecular principles by which opposing family members inhibit, mediate or promote MOM perforation remain elusive. Here, we demonstrate that cell-protecting Bcl-2 directly sequesters cell-killing Bax into a protein-protein complex at the membrane interface preventing Bax forming apoptotic pores.

    By Sebastian Köhler, Sophie E Ayscough, Luke Ashley Clifton, Jörgen Ådén · bioRxiv

    Jan. 27, 2025

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Company Info

Berliner Morgenpost