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Communications Advisor, Ministerio de Sanidad (Spain)
Beats
Beauty,Physical TherapyVitamins & Supplements,WellnessIntensive Care Physical Therapy topics.,LifestyleNews Reporting,Senior CareNutrition & Fitness,Rehabilitation
Reality TV, sports
Content
Glial Alterations in the Glutamatergic and GABAergic Signalling Pathways in a Mouse Model of Lafora Disease, a Severe Form of Progressive Myoclonus Epilepsy - Viana - 2025 - Neuropathology and Applied Neurobiology - Wiley Online Library
By Rosa Okta Viana, Teresa Rubio, Ángela Campos-Rodríguez, Pascual Sanz| Wiley Online Library Verified Conflicts of Interest The authors declare no conflicts of interest. Supporting Information Filename Description nan70009-sup-0001-Suppl-Material.pdfPDF document, 12.6 MB Table S1 List of antibodies used in this work, with reference and commercial source. Table S2 Statistics summary table of the quantification of immunofluorescence and western blot analyses described in this study. Figure S1 Immunofluorescence analyses of different NMDA receptor subunits.
Glial alterations in the glutamatergic and GABAergic signaling pathways in a mouse model of Lafora disease, a severe form of progressive myoclonus epilepsy
By Rosa Okta Viana, Teresa Rubio, Ángela Campos-Rodríguez, Pascual Sanz| bioRxiv@ AbstractLafora disease (LD; OMIM#254780) is a rare form of progressive myoclonus epilepsy characterized by the accumulation of insoluble deposits of aberrant glycogen (polyglucosans), named Lafora bodies (LBs), in the brain but also in peripheral tissues. It is assumed that the accumulation of LBs is related to the appearance of the characteristic pathological features of the disease.
Glial Alterations in the Glutamatergic and GABAergic Signalling Pathways in a Mouse Model of Lafora Disease, a Severe Form of Progressive Myoclonus Epilepsy - Viana - 2025 - Neuropathology and Applied Neurobiology - Wiley Online Library
By Rosa Okta Viana, Teresa Rubio, Ángela Campos-Rodríguez, Pascual Sanz| Wiley Online Library Verified Conflicts of Interest The authors declare no conflicts of interest. Supporting Information Filename Description nan70009-sup-0001-Suppl-Material.pdfPDF document, 12.6 MB Table S1 List of antibodies used in this work, with reference and commercial source. Table S2 Statistics summary table of the quantification of immunofluorescence and western blot analyses described in this study. Figure S1 Immunofluorescence analyses of different NMDA receptor subunits.
Company Info
Ministerio de Sanidad (Spain)